Atherosclerosis Disease Management by Naima Carter-Monroe, Saami K. Yazdani, Elena Ladich, Frank

By Naima Carter-Monroe, Saami K. Yazdani, Elena Ladich, Frank D. Kolodgie, Renu Virmani (auth.), Jasjit S. Suri, Chirinjeev Kathuria, Filippo Molinari (eds.)

Atherosclerosis is a degenerative procedure affecting blood vessels, which determines narrowing of the lumen, plaque development, and hardening of the partitions. it's a probability issue for cardiovascular illnesses. the point of interest of this publication is at the administration of the atherosclerotic disorder. The assurance spans from histological presentation of some of the phases of atherosclerotic lesions to the earliest reviews in atherosclerosis remedy, from complex scientific prognosis to tracking, follow-up, and home-care of the atherosclerotic sufferer. This publication indicates well-established diagnostic innovations protecting numerous scientific imaging modalities similar to Ultrasounds, IVUS, MRI, laptop Tomography, besides new tendencies in early and complex atherosclerosis analysis (innovative medicinal drugs and tissue characterization procedures). Surgical criteria might be provided besides cutting edge experimental trials for the remedy of the atherosclerotic sufferer. The e-book additionally covers rising recommendations in keeping with molecular imaging and vibro-acoustics.

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Radiolabelling autologous monocytes with 111-indium-oxine for reinjection in patients with atherosclerosis. Prog Clin Biol Res 1990;355:271–80. 82. Rudd JH, Warburton EA, Fryer TD, Jones HA, Clark JC, Antoun N, Johnstrom P, Davenport AP, Kirkpatrick PJ, Arch BN, Pickard JD, Weissberg PL. Imaging atherosclerotic 32 N. K. Yazdani, E. D. Kolodgie, and R. Virmani plaque inflammation with [18F]-fluorodeoxyglucose positron emission tomography. Circulation 2002;105:2708–11. 83. Kolodgie FD, Petrov A, Virmani R, Narula N, Verjans JW, Weber DK, Hartung D, Steinmetz N, Vanderheyden JL, Vannan MA, Gold HK, Reutelingsperger CP, Hofstra L, Narula J.

Kolodgie, and R. Virmani and the vasa vasorum. The maximum frequency of hemorrhage is observed in arteries with 50–75% cross-sectional area luminal narrowing [24]. We have reported in coronary plaques that intraplaque hemorrhage is responsible for necrotic core enlargement and excessive foamy macrophages in the fibrous caps [39]. Red blood cell membranes are the richest source of free cholesterol as compared to any other cell in the body. The free cholesterol in the necrotic core is believed to arise from apoptotic cell death of foamy macrophages.

Davies MJ, Thomas AC. Plaque fissuring – the cause of acute myocardial infarction, sudden ischaemic death, and crescendo angina. Br Heart J 1985;53:363–73. 36. Lendon CL, Davies MJ, Born GV, Richardson PD. Atherosclerotic plaque caps are locally weakened when macrophages density is increased. Atherosclerosis 1991;87:87–90. 37. , Kolodgie FD, Piepgras DG, Piperno G, Liotti D, Narula J, Righini P, Ippoliti A, Spagnoli LG. A pathobiologic link between risk factors profile and morphological markers of carotid instability.

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